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Researcher:Professor Peter Berger
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Professor Peter Berger

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Professor Peter BergerResearcher


Institute for Biomedical Ageing Research, Austrian Academy of Sciences (OEAW)

Job title

Group Leader; Professor; Lecturer


Institute for Biomedical Ageing Research
Rennweg 10
6020 Innsbruck


Biology of Ageing / Biogerontology

Specialist areas

Prostate disease

Methodology areas

Molecular biology
Biomarker detection and measurement


Personal Data
Date of Birth: September 16, 1951
Place of Birth: Innsbruck, Tyrol, Austria
Nationality Austrian
1959-1961 American International School in Vienna/Austria
1971 High School Diploma (Albert Schweitzer-Gymnasium) in Hamburg/Germany

Career History
1971-1979 Institute for Microbiology, University of Innsbruck; Research assistent.
1979-1986 Institute for General and Experimental Pathology, University of Innsbruck; PhD, Group leader
1987-1990 Group leader at the Immunoendocrinology Research Unit of the Austrian Academy of Sciences
1989 Visiting Scientist at the European Molecular Biology Laboratories (EMBL) on an EMBO short term fellowship: Epitope analyses of glycoprotein hormones with monoclonal antibodies and natural and synthetic peptides
1990 Visiting Scientist at the Technical University of Vienna on a Kamillo-Eisner research fellowship: Biosensors for Pregnancy- and Tumordiagnosis
1993 Assistant professor at the University of Innsbruck
1990-present Head of the Endocrinology Unit at the Institute for Biomedical Aging Research of the Austrian Academy of Sciences; Major field of interest in research is the “Endocrinology of the Aging Male“
1994-present Institute for Biomedical Aging Research of the Austrian
Academy of Sciences; Deputy Director
1998 Appointed Professor Corresponding member of the Tyrolean Cancer Research Institute (TKFI) in Innsbruck/Austria Corresponding member of the Tyrolean Cancer Research Institute (TKFI) in Innsbruck/Austria

155 Original Publications,
26 Reviews/Book,
numerous invited lectures (>50)

Research Interests
Endocrinology of the Aging Male. The impact of senescence-associated endocrine, molecular and cellular changes paving the way and increasing the susceptibility to prostatic proliferative diseases.